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DocXus retweeted
This is NOT CPR
Every time a clip like this goes viral, thousands of people believe that chest pain = start chest compressions
It doesnât
In fact, doing CPR on someone whose heart is still beating can do more harm than good
CPR is not the treatment for chest pain
CPR is ONLY for CARDIAC ARREST
You start CPR ONLY when the person is:
1. UNRESPONSIVE (Unconscious, won't wake up)
2. NOT BREATHING (or only gasping)
3. PULSELESS (No heartbeat)
News channels and viral social media pages continue to circulate videos like this without any medical context
Millions watch them, save them, and assume this is exactly what they should do in an emergency
If someone develops severe chest pain, seek urgent medical care. Do not start chest compressions
CPR is reserved for when the person collapses into cardiac arrest
Please donât learn CPR from viral clips.
Learn it from certified courses or reliable educational videos.
Knowing when not to perform CPR is just as important as knowing how to perform it.
Chhatrapati Sambhajinagar - A woman passenger suffered a heart attack at the bus stand in Sambhajinagar, but was saved by the quick thinking of woman conductor Sangita Palwe.Palwe immediately administered CPR to the passenger. Despite calling 108, the ambulance did not arrive on time. The woman was then taken to the hospital in a rickshaw and admitted for treatment.The conductorâs presence of mind and timely action are being widely praised for saving the passengerâs life.
DocXus retweeted
This didnât have to happen
And it wonât, if you read the next 2 minutes carefully
A dog bite does NOT automatically mean rabies.
Rabies only transmits if the dog biting you is already infected with the virus. Thatâs it. Thatâs the only way
A rabid dog isnât just âaggressive.â Itâs a different animal entirely. Foaming at the mouth
Biting everything in sight - people, other animals, objects. Disoriented. Erratic. Visibly, unmistakably sick
A scared stray that bit you when you got too close is almost certainly not rabid
But hereâs where people make the fatal mistake:
They think âprobably not rabidâ means âI donât need a vaccineâ
Wrong
Because when rabies actually develops, it is 100% fatal. No treatment. No second chance. Game over.
The vaccine exists precisely for this uncertainty. And it works spectacularly
If you get bitten. Do this. In order. No shortcuts
Step 1 - Wash immediately.
Not a quick rinse. Soap and running water, hard, for 15 full minutes. This one step alone can flush the virus before it even enters your system
Step 2 - Go to a hospital today.
Not tomorrow. Not after you âsee how it looks.â Today
Step 3 - Get your PEP vaccines.
Post-Exposure Prophylaxis. Four shots over 14 days (Day 0, 3, 7, 14). Close to 100% effective when started on time. This is the science working exactly as intended
Step 4 - Deep bite? Face or neck? Get RIG too
Rabies Immunoglobulin is injected directly at the wound site - itâs your bodyâs immediate shield while the vaccine builds long-term protection. Your doctor will assess this
Step 5 - Watch the dog if you can
A dog thatâs alive and healthy 10 days after biting you was almost certainly not rabid. This is a recognised clinical rule
The brutal truth of this tragedy:
Modern medicine had already solved this problem
The vaccine exists. The immunoglobulin exists. The protocol is simple, accessible, and nearly foolproof.
Share this post. Bookmark it. Send it to someone
Because somewhere right now, someone just got bitten by a dog, and theyâre spiralling into the same fear
They donât need to.
Man, 30, Dies By Suicide. He Thought He Would Contract Rabies After Dog Bite
ndtv.com/india-news/man-30-dâŠ
DocXus retweeted
A new allergy is spreading across the US and most doctors have never heard of it
A 47 year old walked into the ER in feeling faint with swollen eyelids, an itchy rash, and 3 weeks of watery diarrhea after every meal.
For the past 5 days the diarrhea had progressed to crampy abdominal pain, nausea, and vomiting.
His HR was high. BP was low. Hives across his scalp, neck, shoulders, abdomen, and groin. Lips swelling.
Then his blood pressure crashed.
> white cell count massively elevated
> lactic acid high indicating poor tissue perfusion
> troponin elevated suggesting cardiac stress
> procalcitonin elevated suggesting possible infection
> kidney function declining
They pumped 4 litres of IV fluids. Blood pressure came back. Heart rate stayed high.
Then it crashed again.
2 more litres. Antibiotics. H2 blockers. Glucocorticoids. Then 10 more litres. He was moved to the ICU on epinephrine.
Blood and urine cultures came back negative. Not sepsis.
By day 3 he was improving. Rash fading. Pain settling.
Then on day 4 it all came back. Chest tightness. Difficulty breathing. Worsening rash. Tachycardia. Hypotension. Oxygen dropping. Wheezing.
Epinephrine rescued him again almost immediately.
His bloodwork from day 1 finally returned. Elevated IgE. Elevated tryptase. This was mast cell driven anaphylaxis.
Something was triggering a massive allergic response. But to what?
He had no new medications. No new foods. No known allergies. No travel.
Then someone asked what he ate 4 hours before the day 4 flare.
Beef
He also mentioned he had eaten venison (meat from a deer) 2 days before the original ER visit. And that he is a deer hunter who had recently been hunting white tailed deer in southeastern Michigan.
He had alpha-gal syndrome.
Alpha-gal is a carbohydrate found in all nonprimate mammals.
Humans donât naturally have it.
But if you get bitten by a lone star tick that has previously fed on a deer or other mammal the tickâs saliva introduces alpha-gal into your bloodstream.
Your immune system produces IgE antibodies against it.
And now every time you eat red meat your body treats it as a foreign invader and launches a full allergic response.
The reaction is delayed by 3 to 6 hours after eating which is why itâs so hard to connect to food. Most allergies hit within minutes. Alpha-gal waits. That delay is what made this case so difficult to crack.
The lone star tick used to be confined to the eastern and southeastern United States.
It has now spread into the upper Midwest, the Northeast, and eastern Canada.
The cause is climate change. Warmer temperatures. Shifting precipitation. Expanding habitats for both the tick and the white tailed deer it feeds on.
Alpha-gal syndrome cases in the US have surged from near zero to over 110,000 suspected cases in the last decade.
Most physicians have never seen one. Many donât even know it exists
Climate change isnât just about rising sea levels and wildfires.
Itâs changing which diseases show up in your doctorâs office.
And if your doctor isnât thinking about it you might end up in the ICU for eating a BURGER
DocXus retweeted
An example of inadequate history takingâŠ
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup couldâve been replaced by a 5 minute conversation about food.
DocXus retweeted
OdliÄan tekst o (relativno teĆĄkoj) dijagnozi skorbuta u moderno vreme
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup couldâve been replaced by a 5 minute conversation about food.
DocXus retweeted
I just watched a whole new ep of House MD
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup couldâve been replaced by a 5 minute conversation about food.
DocXus retweeted
But I hear all the time on X, "We are tired of hearing doctors drone on about our diet and lifestyle. It's patronizing and minimizing."
This is why we start the conversation with diet. đ
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup couldâve been replaced by a 5 minute conversation about food.
DocXus retweeted
Hace tiempo lleguĂ© a la guardia de sĂĄbado, en el pase de visita vimos a una chica de 23 años con ascitis ++, sin ictericia, edema bimaleolar ++, piel acartonada, Hb 6.5g, hepatomegalia, no hemorragia, Dx presuntivo "Probable CA hepĂĄtico, a descartar hepatitis autoinmune (?). đ§”
A 2 year old in the US was diagnosed with a disease from the 1700s
He was brought to the ER because he stopped walking.
No trauma. No fall. He just slowly stopped bearing weight on his legs over 6 weeks until he was crawling instead of walking.
His gums were swollen and bleeding. He had a rash. Pinpoint bleeding under his skin. Pain in both legs. Decreased muscle tone.
He kept his legs in a frog leg position and screamed when anyone touched them.
The doctors suspected everything.
First visit: possible fracture. Orthopedics recommended repeat imaging. Four weeks later he was worse.
Second visit: concern for leukemia. Then concern for nonaccidental trauma. Child protective services was called.
Then the workup began
> CT scan of both legs. No fracture.
> MRI of head and spine. Normal.
> lumbar puncture to rule out Guillain Barre. Normal.
> inflammatory markers. Normal.
> muscle enzymes. Normal.
> heavy metal screening. Negative.
> vitamin D and B12. Normal.
> bone marrow biopsy to rule out cancer. Normal.
> electromyography. Normal.
> muscle biopsy. Normal.
Weeks of testing. Multiple hospital transfers. Invasive procedures on a toddler. His parents watched their child get poked and scanned and biopsied while nobody could tell them what was wrong.
Then someone asked the right question.
What does he eat?
Chocolate milk and graham crackers. That was his entire diet. His mother had mentioned he was a picky eater at the very first visit. Nobody followed up on it.
His vitamin C level came back at less than 0.1 mg per deciliter.
He had scurvy.
The same disease that wiped out entire ship crews during the Age of Exploration. The same disease the British Navy solved in 1795 by issuing lemon juice to sailors.
Scurvy impairs collagen production and compromises blood vessel integrity. That explains the bleeding gums. The rash. The periosteal elevation on MRI. The bone pain. The inability to walk.
Every single symptom pointed to one thing and it took months to get there.
He was started on vitamin C supplements. Within a week he was moving his legs again. At 4 months he was walking and his hemoglobin was normal.
The entire workup could have been avoided with one detailed dietary history at the first visit.
We have MRIs and bone marrow biopsies and genetic panels. But the most powerful diagnostic tool in medicine is still asking a parent what their child eats for dinner.
All that extensive workup couldâve been replaced by a 5 minute conversation about food.